Free Radicals Test
A - Z of Health
Research
Therapy Overviews
Health Guider
Health Insurance
Case Studies
Contributors
Support Groups
Professional Bodies

Thought For Today
health insurance
metallic taste
 
 


Members Area   :   Update your details   |   Lost password   |   Discussion groups
Practitioner Search Health Centre Search Advertise With Us Join Our Directories

OSTEOPATHY and Lower Back Pain

By Laurence Kirk

Patient: Ms A., a 35yr old female equestrian height 5ft 4ins weight 10stone

Complaint: Acute low back pain of two weeks duration as a result of mucking
out a stable whilst on holiday.

History
Ms A had suffered acute recurrent low back pain from the age of 19
years after falling from a horse (X-rays at the time revealed no fractures).

From that time she developed slight weakness and hypoaesthesia of the
entire left lower leg diagnosed by a neurologist as "stocking syndrome"
When she became more active with her equestrian career and was riding
regularly she had few problems although she was always aware of the reduced
sensation in her left leg.

Her current pain was described as continuous and sharp in nature and
localised to the left lower back, there was some referral of pain into the
posterior left thigh accompanied by pins and needles into the left lower.
The pain was generally aggravated by activity by changes in position,
sitting for long periods, and there was impulse pain on coughing or
sneezing. There was no morning stiffness reported

Ms A was generally stoic by nature and had developed effective coping
strategies over the years to allow her to work through her discomfort and
continue to compete at a relatively high level, unfortunately the recent
exacerbation had proved to be one straw too many.

Examination
Standing examination revealed an asymmetry of posture with an
elevated left ileum and mild lumbar structural scoliosis concave to the
left. On palpation the lumbar erector spinae were hypertonic and tender with
focal areas of tenderness over the left iliac crest and psis. The left lower
limb appeared to be slightly longer than the right (<1.5cm). Range of active
motion was reduced and painful in the lumbar spine for flexion and left
rotation/side bending. Restriction of motion and tenderness was also noted
in the cervical spine.

Neurological examination was somewhat curious; there
appeared to be complete anaesthesia of the entire left lower limb other than
the medial border of the foot. Reflexes were normal and equal but the left
lower limb showed generally reduced muscle strength. Neural tension tests
interestingly were all negative (although these tests are acknowledged as
being somewhat unreliable) percussion was unremarkable. Peripheral pulses
were slightly reduced on the left. Provocative testing for sacroiliac
involvement appeared positive on the left.

Treatment
Ms A had already seen her GP and had been prescribed diazepam
and ibuprofen. Treatment was directed to restore a degree of function to the left S/I joint and relieve the acute pain associated with this. Gentle conservative
approaches were used given the severity of discomfort. This took the form of
rhythmic oscillatory mobilisation of the S/I and lumbar spine and soft
tissue treatment of the involved musculature. A knee swinging exercise was
recommended to improve proprioceptive ability in the lower back

After the first treatment 5 days later Ms A noted a significant increase in
mobility and reduction in pain. Of more interest was the observation that
slight sensations had been noted in the previously anaesthetic left lower
leg. On testing the L5 dermatome exhibited some sensitivity to both light
touch, and vibration.

On the third visit Ms A felt very much improved and extremely grateful that
sensation in her left leg after 16 years was now all but back to normal
(1 year follow up indicated that the improvement had been maintained).

Was the actual treatment responsible for the resolution of her long term
paraesthesia? Could the strenuous exertion of mucking out the stable and
moving into awkward postures have triggered off some recovery mechanism? Is
it likely that nerves which have been dysfunctional for 16 years can recover
in a matter of weeks? Was her problem vascular rather than neurological,
mediated via the sympathetic outflow to the lower limb rather than somatic
innervation?

Case Courtesy of
Laurence Kirk

THE BRITISH COLLEGE OF NATUROPATHY AND OSTEOPATHY
Lief House
3 Sumpter Close
120-122 Finchley Road
London NW3 5HR
Tel: 020 7435 6464
Fax: 020 7431 3630

Details of training can be obtained from the Registrar at the above address
There is an out-patient clinic at the College
For further details and appointments, telephone 020 7435 7830

Related Books
Practitoners Search
Health Centre Search
Sitemap  |  Disclaimer  |  Developed by moragan